Paolo Desenzani @despal65
Medico Chirurgo Specialista in Endocrinologia e Diabetologia, Geriatria paolodesenzani.it Joined September 2012-
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¿Qué tiempos estamos viviendo? A mí me tocó cuando la diabetes se dividía en insulinodependiente y no insulinodependiente; después llegó la era tipo 1 y tipo 2; luego aparecieron las formas específicas (MODY, LADA, diabetes pancreática), y ahora el debate se ha desplazado hacia el otro extremo del espectro: la prediabetes. Un grupo de especialistas publicó recientemente en Lancet Diabetes & Endocrinology una propuesta de Shah VN et al. para eliminar el término "prediabetes" y sustituirlo por una clasificación en tres etapas de diabetes tipo 2, adoptando el término "disglucemia" para las etapas previas a la enfermedad manifiesta. El argumento de fondo no es menor: solo un tercio de los pacientes etiquetados como prediabéticos desarrolla diabetes; el resto permanece en disglucemia o revierte a la normalidad. Y mientras tanto, la palabra "prediabetes" transmite, casi inevitablemente, la idea de que todavía se está sano. No existe un proceso de aprobación farmacológica formal para esta condición, aunque agentes como metformina, pioglitazona y los agonistas del GLP-1 han demostrado que pueden reducir la progresión hacia la enfermedad manifiesta. La clasificación propuesta quedaría así: etapa 1 (glucemia normal, con glucosa en ayunas en ascenso pero dentro del rango normal, HbA1c menor de 5.7% y TITR mayor de 90-95%); etapa 2 (disglucemia, con glucosa por encima de lo normal sin alcanzar criterios diagnósticos de DM2, HbA1c entre 5.7 y 6.4%, lo que hoy conocemos como prediabetes), subdividida en 2a para progresores lentos y 2b para progresores rápidos; y etapa 3 (DM2 manifiesta, con glucosa en ayunas mayor o igual a 7.0 mmol/L, HbA1c mayor o igual a 6.5% y TITR menor de 80%). Y aquí es donde la etapa 1 cobra una relevancia que muchas veces pasamos por alto, aunque también es la más difícil de delimitar: un paciente puede llevar años con glucosas en ayunas entre 90 y 105 mg/dL (técnicamente normales) y al mismo tiempo tener un HOMA-IR de 8, con el páncreas trabajando a marchas forzadas para mantener esa apariencia de normalidad. Es la luna de miel metabólica: la glucemia luce tranquila en el laboratorio, pero detrás de esa fachada hay una resistencia a la insulina que ya está instalada y una célula beta que está pagando el precio. Cuando el páncreas ya no pueda compensar, la glucosa subirá, y para ese momento, el daño lleva años en curso. Vale añadir que el riesgo cardiovascular en estas etapas tempranas parece relacionarse tanto con el síndrome metabólico en su conjunto como con la hiperglucemia per se, lo que complica aún más la interpretación clínica de la etapa 1. Vale la pena aclararlo antes de seguir: esto no es una guía vigente. La ADA 2026 conserva el término prediabetes. Se trata de una propuesta académica con defensores y con críticos, y el debate apenas comienza.
Il «fegato grasso» fa male anche al cuore: un problema serio che non dà sintomi. Ecco a che cosa dar peso corriere.it/salute/nutrizi…
Grande onore aver conosciuto dr ssa Melanie Davies diabetologa UK primo nome delle Linee Guida diabetologiche ADA EASD !!!!!
Semaglutide: una nuova opzione terapeutica per lo scompenso a frazione d’eiezione preservata cardiolink.it/abstract/?id=1…
Lo psicologo più importante del mondo rivela 21 regole che cambieranno la tua vita in meglio curiosandosimpara.com/2019/01/09/lo-…
Prevenire il diabete «in famiglia»: anticipare l’orario della cena può essere utile? corriere.it/salute/nutrizi…
Just 4,000 daily steps may lower your risk of death, study finds nbcnews.com/health/health-… via @nbcnews
Dai un'occhiata al video di paolodesenzani! #TikTok vm.tiktok.com/ZGJxJ1k3D/?t=2
Dai un'occhiata al video di paolodesenzani! #TikTok vm.tiktok.com/ZGJ9NNrb6/?t=2
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